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By Robert Plutchik

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Cortisol) does not necessarily increase in the pure fight pattern of response. When the feeling of fear-anxiety predominates, the response involves basal amygdalar activation with a predominance of epinephrine. , 1978). This is, in part, because there is a decrease in resistance in the vascular bed of the muscles as a result of activation of the cholinergic system. the overall response of an anxious animal is one of a sudden, intense explosion of muscular effort directed towards flight with increased cardiac output.

Weber, J. , Hogan, C , & Price, T. D. (1954). Metabolic changes following destructive lesions in the forebrain in cats. In R. G. ), Studies in schizophrenia, (pp. 61-81). Cambridge, Massachusetts: Harvard University Press. Heilman, K. , & Watson, R. T. (1975). Auditory affective agnosia. Disturbed comprehension of affective speech. Journal of Neurology, Neurosurgery, & Psychiatry, 38, 69-72. Jones, E. , Saper, C. , & Swanson, L. W. (1976). Midbrain, diencephalic and cortical relationships of the basal nucleus of Meynert and associated structures in primates.

But when clinical seizures began, the abnormal activity involved the cortex, just as with seizures in patients. These studies showed that the deep cerebellar nuclei participated in the propagation of the seizural discharge in a consistent relationship with other structures within the network for emotion. ) The temporal relationship of the cerebellar activity was consistent and unique in that seizural activity did not appear in the cerebellar nuclei until the clinical seizure was well under way, involving all supratentorial sites.

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